CHAMGAP
APPROVEDReviewed and approved by the Chamgap Editorial Team (2026-07-21). The draft was written by AI, the existence of all 3 cited sources was verified at the original page, and the verdict passed blind grading and adversarial audit. Methodology v0.6.
Verdict No. 1018 · Search date 2026-07-21 · Methodology v0.6

SAMe,
does it really help with Reduced risk of death or liver transplantation in alcoholic cirrhosis?

30-Second Summary
C
Evidence Grade C · 41 · Safety caution
The overall reduction in death or transplantation was nonsignificant, with only a less-advanced-disease subgroup leaving a possibility signal
What the
research shows
SAMe is rated at the bottom of C for death or liver transplantation in alcoholic cirrhosis. The pivotal 123-person, two-year trial found a large direct hard-outcome signal, with death or transplantation of 30% on placebo versus 16% on SAMe, but the primary endpoint was nonsignificant at p=0.077 and the study was underpowered. The 29% versus 12%, p=0.025 result after excluding eight Child class C participants was a subgroup analysis, not confirmation. The Cochrane death-or-transplant estimate of RR 0.55 with a 95% CI of 0.27 to 1.09 was imprecise, spanning a large benefit through no effect, and no independent confirmatory trial exists. Under the rule ① ceiling for a null primary endpoint, the large estimate and hard-outcome directness support C with 41 points.
What the
ads claim
Marketing turns methyl donation and glutathione synthesis into claims of liver regeneration, longer survival in cirrhosis, and delayed transplantation. The pivotal clinical trial produced an encouraging numerical difference but a statistically nonsignificant overall endpoint, and a mechanism or enzyme change is not the same as fewer deaths or transplants.
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Useful facts when choosing a product

  • The pivotal alcoholic-cirrhosis trial used oral SAMe 1,200 mg per day for two years but did not obtain a statistically significant overall death-or-transplant result.
  • SAMe can be unstable with heat and moisture, and salt form, enteric coating, storage, and actual content vary among supplements, so the trial formulation cannot represent every product.
  • Alcohol abstinence, nutrition, prevention and treatment of cirrhosis complications, liver-cancer surveillance, and transplant evaluation remain the core of survival-oriented care and must not be replaced by SAMe.
  • Usually mild nausea, abdominal discomfort, insomnia, or anxiety can occur, while people with bipolar disorder face a rare risk of mania or hypomania and should review combinations with serotonergic medicines with a clinician.
Gap Measurement · Verdict 1018 · C 41
What advertising claims
What independent, higher-quality research supports
△ GAP
01

What the research actually shows

Mato and colleagues enrolled 123 patients with alcoholic cirrhosis, with histological confirmation in 84%, including 75 in Child class A, 40 in B, and eight in C, and treated them in a multicenter double-blind trial for two years. Death or transplantation in the full analysis was 30% versus 16%, a large difference, but the primary endpoint was nonsignificant at p=0.077 and the trial was underpowered. Excluding Child class C produced 29% versus 12%, p=0.025, with survival-curve p=0.046, but this was a subgroup analysis. The Cochrane death-or-transplant estimate was RR 0.55 with a 95% CI of 0.27 to 1.09, spanning large benefit through no effect. The later 37-person trial was null and not sized for death or transplantation, and no large independent confirmatory trial exists.

02

Why this is classified as C (41)

The direct pivotal trial showed a large hard-outcome estimate of 30% versus 16% death or transplantation, but the primary result was nonsignificant at p=0.077 and underpowered. The Cochrane RR of 0.55 had an imprecise 95% CI of 0.27 to 1.09, the Child class C-excluded result was an unconfirmed subgroup analysis, and no independent confirmation exists. The null-primary-endpoint ceiling applies, but the limited direct hard-outcome signal supports bottom-of-C with 41 points rather than D. Product variation and adverse effects remain separate.

Counterpoint. A real effect in less advanced Child class A or B disease remains possible. It would require a sufficiently large, prospectively stratified trial on top of modern standard care that tests death, transplantation, and cirrhosis complications; current evidence does not support taking SAMe for a survival benefit.

Rejudgment record. Cross-check incorporated — Applied the rule ① ceiling of C because the 123-person, two-year trial produced a large direct death-or-transplant estimate of 30% versus 16% but the primary endpoint was nonsignificant at p=0.077 and underpowered; the Child class C-excluded result was a subgroup analysis, the Cochrane RR of 0.55 had an imprecise 95% CI of 0.27 to 1.09, and no independent confirmatory trial exists. The limited hard-outcome signal supports the bottom of C rather than D

Sub-claim grades by effect

This ingredient is marketed for several effects. A single overall grade blends strong and weak claims together, so each effect is graded separately here. The overall grade reflects the strongest disconfirming or core claim.

Effect (sub-claim)GradeBasis
Reduced death or liver transplantation in the full alcoholic-cirrhosis populationCThe pivotal 123-person, two-year trial showed a large hard-outcome signal of 30% versus 16%, but the primary endpoint was nonsignificant at p=0.077, underpowered, and unconfirmed independently.
Reduced all-cause mortality in alcoholic cirrhosisCThe Cochrane all-cause mortality estimate was RR 0.62 with an imprecise 95% CI of 0.30 to 1.26, spanning large benefit through no effect, without a confirmatory trial.
Reduced death or transplantation in Child class A or B alcoholic cirrhosisCExcluding eight Child class C participants produced 29% versus 12%, p=0.025, but this was a subgroup analysis without independent confirmation.

Cross-check — Codex and Claude

This verdict was drafted by Codex through literature review and source-existence checks, cross-checked through blind grading and adversarial audit, and settled by reapplying the methodology boundary rules. Cases with split grades were resolved through rejudgment.
03

Evidence Table

StudyDesignSampleFundingEndpointResultWeight
Mato JM et al. 1999Multicenter randomized double-blind placebo-controlled trial2Funding source not reported in the PubMed abstractAll-cause death or liver transplantationThe result was 30% with placebo versus 16% with SAMe but nonsignificant at p=0.077; only exclusion of Child class C produced 29% versus 12%, p=0.025.Pivotal direct hard outcome with a large estimate but a nonsignificant, underpowered primary analysis
Rambaldi A, Gluud C. 2006Cochrane systematic review and meta-analysis of randomized trials434Academic Cochrane Hepato-Biliary Group reviewAll-cause mortality, liver-related mortality, death or transplantation, and complicationsBoth all-cause mortality RR 0.62 and death-or-transplant RR 0.55 had confidence intervals crossing no effect.Imprecise pooled estimate without confirmation
Medici V et al. 2011Randomized double-blind placebo-controlled trial24Funding source not reported in the PubMed abstractLiver enzymes, clinical and biochemical measures, and liver histologyThe cohort improved with abstinence, but no clinical, biochemical, or histological difference appeared between SAMe and placebo.Later small null trial
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Receipt — 3 References

All 3 cited sources were verified for existence at the original page (as of 2026-07-21).

Mato JM, Cámara J, Fernández de Paz J, et al. S-adenosylmethionine in alcoholic liver cirrhosis: a randomized, placebo-controlled, double-blind, multicenter clinical trial. J Hepatol. 1999;30(6):1081-1089. PMID: 10406187. DOI: 10.1016/S0168-8278(99)80263-3.
checked
Rambaldi A, Gluud C. S-adenosyl-L-methionine for alcoholic liver diseases. Cochrane Database Syst Rev. 2006;2006(2):CD002235. PMID: 16625556. DOI: 10.1002/14651858.CD002235.pub2.
checked
Medici V, Virata MC, Peerson JM, et al. S-adenosyl-L-methionine treatment for alcoholic liver disease: a double-blinded, randomized, placebo-controlled trial. Alcohol Clin Exp Res. 2011;35(11):1960-1965. PMID: 22044287. PMCID: PMC3315189. DOI: 10.1111/j.1530-0277.2011.01547.x.
checked
Draft and rewrite: Codex (AI) · Verification: Codex blind grading and adversarial audit · Final adjudication: Claude
Reviewed and approved: Chamgap Editorial Team · Approval date: 2026-07-21 · Corrections: none

Cite this verdict

SAMe x reduced death or liver transplantation in alcoholic cirrhosis Evidence Grade C card
[Chamgap] SAMe x reduced death or liver transplantation in alcoholic cirrhosis — Evidence Grade C·41. 3 cited sources checked. Source: https://chamgap.com/en/verdicts/liver/same-alcoholic-cirrhosis-death-liver-transplantation/ · CC BY 4.0

CC BY 4.0 — free to use with attribution; do not distort grades, numbers, or verdict meaning.

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What this document does and does not do

Chamgap is an information source. It reports what research has and has not confirmed; it does not tell readers what to take or buy. That decision belongs to readers and, when needed, medical or legal professionals. This verdict reflects literature available up to the search date and may change as new research appears. Nothing here is medical advice.