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APPROVEDReviewed and approved by the Chamgap Editorial Team (2026-07-24). The draft was written by AI, the existence of all 5 cited sources was verified at the original page, and the verdict passed blind grading and adversarial audit. Methodology v0.6.
Verdict No. 1831 · Search date 2026-07-24 · Methodology v0.6

Early infant peanut introduction,
does it really help with Prevention of peanut allergy in infants?

30-Second Summary
A
Evidence Grade A · 90 · Safety caution
Early infant peanut introduction prevents peanut allergy, especially in high-risk infants, but does not treat established allergy
An allergic reaction can occur at first ingestion, and whole peanuts create a choking hazard. Infants with severe eczema, egg allergy, or suspected sensitization may need clinician-guided introduction and an age-appropriate form.
What the
research shows
LEAP introduced peanut at age 4 to 11 months in high-risk infants and continued regular intake to age five, producing a large reduction in challenge-confirmed peanut allergy and an A grade. Among 640 randomized infants, the evaluable intention-to-treat population of 628 had overall prevalence of 17.2% with avoidance versus 3.2% with consumption. The requested figure of 13.7% is not the overall avoidance rate; it belongs to the baseline skin-test-negative cohort, whose corresponding consumption rate was 1.9%. A meta-analysis of four trials and 3,796 participants, including EAT, independently replicated benefit with RR 0.31. LEAP enrolled infants with severe eczema or egg allergy, and this is prevention rather than treatment of established allergy.
What the
ads claim
Marketing can simplify the evidence into giving peanut casually to every infant or imply that established allergy disappears. The evidence concerns prevention with age-appropriate forms and regular intake; high-risk infants may require supervised assessment before introduction.
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Useful facts when choosing a product

  • LEAP used at least 6 g of peanut protein per week in three or more meals from age 4 to 11 months through 60 months; whole peanuts were not used because they are a choking hazard.
  • The overall evaluable intention-to-treat comparison was 17.2% versus 3.2%; the baseline skin-test-negative cohort was 13.7% versus 1.9%, and the positive cohort was 35.3% versus 10.6%.
  • Verdict 1772, which is C with 58 points, concerns omalizumab treatment to increase tolerated protein amounts in patients who already have multiple food allergies. This verdict concerns prevention before peanut allergy develops.
Gap Measurement · Verdict 1831 · A 90
What advertising claims
What independent, higher-quality research supports
△ GAP
01

What the research actually shows

LEAP randomized 640 infants aged 4 to 11 months who had severe eczema or egg allergy to peanut consumption or avoidance. Among 628 in the evaluable intention-to-treat population, challenge-confirmed peanut allergy at 60 months was 17.2% versus 3.2% overall, meeting the primary endpoint. Rates were 13.7% versus 1.9% among 530 with a negative baseline skin test and 35.3% versus 10.6% among 98 with a positive test. Logan 2023 evaluated individual data from 1,796 LEAP and general-risk EAT children and found a 75% intention-to-treat reduction. A 2023 JAMA Pediatrics meta-analysis of four randomized trials and 3,796 participants reported RR 0.31 (95% CI 0.19 to 0.51; I-squared 21%) with high certainty. LEAP-On enrolled 556 of 628 eligible LEAP children and analyzed 550 at 72 months, finding persistence after one year of avoidance, 18.6% versus 4.8%; it was follow-up rather than a new randomized replication.

02

Why this is classified as A (90)

A challenge-confirmed event endpoint, 628 evaluable intention-to-treat participants, and an overall 17.2% versus 3.2% result were followed by independent randomized synthesis with RR 0.31. H, R2, I2, E+, and B0 yield A with 90 points.

Counterpoint. Families of high-risk infants should discuss timing, initial testing, and supervision with a pediatric or allergy clinician. A prevention protocol should not be used as treatment for a child with confirmed peanut allergy.

Rejudgment record. Cross-check applied — A challenge-confirmed allergy event fell substantially in a publicly funded pivotal trial and independent randomized synthesis, giving H, R2, I2, E+, and B0

Scoring profile behind this grade
EndpointHHard endpoint - actual events such as death
ReplicationR2Independently replicated across trials
IndependenceI2Decisive evidence is publicly or non-profit funded
Effect sizeE+Meets the clinically important threshold

The scoring table and the verdict agree (A).

Sub-claim grades by effect

This ingredient is marketed for several effects. A single overall grade blends strong and weak claims together, so each effect is graded separately here. The overall grade reflects the strongest disconfirming or core claim.

Effect (sub-claim)GradeBasis
Prevention of peanut allergy by age five in high-risk infants with severe eczema or egg allergyAThe food-challenge primary endpoint improved substantially among 628 evaluable intention-to-treat participants in LEAP.
Prevention of peanut allergy across infants including those at general riskBEAT alone missed its composite intention-to-treat primary endpoint, but pooled LEAP-EAT intention-to-treat and four-trial meta-analysis were positive.
Persistence of preventive benefit for one year after early intake stopsBThe difference persisted among 550 children in LEAP-On, but this was follow-up of the original randomized groups.

Cross-check — Codex and Claude

This verdict was drafted by Codex through literature review and source-existence checks, cross-checked through blind grading and adversarial audit, and settled by reapplying the methodology boundary rules. Cases with split grades were resolved through rejudgment.
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Evidence Table

StudyDesignSampleFundingEndpointResultWeight
Du Toit G et al. 2015 LEAPOpen-label randomized controlled trial with food-challenge ascertainment at 60 months98Public NIAID/NIH funding plus nonprofit Food Allergy Research and Education and MRC/Asthma UK support; peanut supplied by the National Peanut BoardPrevalence of challenge-confirmed peanut allergy at 60 monthsOverall 17.2% versus 3.2%, P<0.001, meeting the primary endpoint; 13.7% versus 1.9% in the negative cohort and 35.3% versus 10.6% in the positive cohort.Pivotal large objective-event trial
Logan K et al. 2023Individual-participant pooled analysis of the LEAP and EAT randomized trials1,796European Union iFAAM public funding; parent trials had public and nonprofit support including NIAID and the UK Food Standards Agency, with some author industry conflicts disclosedFood-challenge-based peanut allergy at age 3 to 5 yearsPooled intention-to-treat analysis showed a 75% reduction, with protection across eczema-severity strata.Replication across broader risk strata
Perkin MR et al. 2016 EATRandomized early-allergenic-food-introduction trial in general-risk infants1,303Public funding from the United Kingdom Food Standards Agency and Medical Research Council plus nonprofit supportAllergy to one or more of six foods through age three yearsThe composite intention-to-treat primary endpoint was null at 7.1% versus 5.6%, P=0.32, with low adherence; the independent trial contributed peanut-specific data to meta-analysis.Independent randomized evidence in general-risk infants
Scarpone R et al. 2023Systematic review and meta-analysis of randomized allergenic-food-introduction trials3,796Peer-reviewed academic synthesis assessing funding sources and risk of bias in included trialsDevelopment of peanut allergyRR 0.31 (95% CI 0.19 to 0.51), I-squared 21%, a high-certainty reduction.Independent multiple-trial synthesis supporting R2
Du Toit G et al. 2016 LEAP-OnTwelve-month peanut-avoidance follow-up of the original LEAP assignments1Public NIAID/NIH funding and nonprofit FARE and MRC/Asthma UK supportChallenge-confirmed peanut allergy at 72 monthsThe difference persisted at 18.6% versus 4.8%, P<0.001; increase from month 60 within the consumption group was not significant.Durability evidence, not independent replication
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Receipt — 5 References

All 5 cited sources were verified for existence at the original page (as of 2026-07-24).

Du Toit G, Roberts G, Sayre PH, et al. Randomized Trial of Peanut Consumption in Infants at Risk for Peanut Allergy. N Engl J Med. 2015;372(9):803-813. PMID: 25705822. DOI: 10.1056/NEJMoa1414850.
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Logan K, Bahnson HT, Ylescupidez A, et al. Early introduction of peanut reduces peanut allergy across risk groups in pooled and causal inference analyses. Allergy. 2023;78(5):1307-1318. PMID: 36435990. DOI: 10.1111/all.15597.
checked
Perkin MR, Logan K, Tseng A, et al. Randomized Trial of Introduction of Allergenic Foods in Breast-Fed Infants. N Engl J Med. 2016;374(18):1733-1743. PMID: 26943128. DOI: 10.1056/NEJMoa1514210.
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Scarpone R, Kimkool P, Ierodiakonou D, et al. Timing of Allergenic Food Introduction to the Infant Diet and Risk of Allergic or Autoimmune Disease: A Systematic Review and Meta-analysis. JAMA Pediatr. 2023;177(5):489-497. PMID: 36972063. DOI: 10.1001/jamapediatrics.2023.0142.
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Du Toit G, Sayre PH, Roberts G, et al. Effect of Avoidance on Peanut Allergy after Early Peanut Consumption. N Engl J Med. 2016;374(15):1435-1443. PMID: 26942922. DOI: 10.1056/NEJMoa1514209.
checked
Draft and rewrite: Codex (AI) · Verification: Codex blind grading and adversarial audit · Final adjudication: Claude
Reviewed and approved: Chamgap Editorial Team · Approval date: 2026-07-24 · Corrections: none

Cite this verdict

Early infant peanut introduction x prevention of peanut allergy Evidence Grade A card
[Chamgap] Early infant peanut introduction x prevention of peanut allergy — Evidence Grade A·90. 5 cited sources checked. Source: https://chamgap.com/en/verdicts/immunity/early-peanut-introduction-infant-peanut-allergy-prevention/ · CC BY 4.0

CC BY 4.0 — free to use with attribution; do not distort grades, numbers, or verdict meaning.

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